AO1 AFFH
An attempt at explaining anorexia nervosa (AN) has been
explained by Guisinger’s (2003) adapted to flee famine hypothesis (AFFH). AFFH
fits under the evolutionary approach, but is also a biological explanation.
Guisinger proposes that AN is part of behaviours adaptive in the environment of
the evolutionary adaptation (EEA). In hunter-gatherer society, there would have
been prolonged periods of famine and it would have been necessary to move on
regularly when food resources were exhausted. To cope with this, people would
have keep active despite lack of energy. Guisinger draws a parallel with
anorexics, who tend to have high activity levels and restlessness, despite the
typical response to starvation being inactivity and depression. Therefore, she
hypothesised, high levels of activity and a denial of hunger, necessary to
migrate in times of famine, and are explanation of AN today.
AO2
Guisinger’s AFFH meets criticism. As
with most research in the evolutionary approach, it is limited by its lack of
direct evidence. AFFH remains a hypothesis, because it cannot be proven.
Guisinger draws from her knowledge and assumptions regarding hunter-gatherer
society and applies them to traits seen in sufferers of AN. Whilst there appears
to be something in what she says, it is has very little scientific evidence to
support it.
AO2/IDA
Another criticism of this theory is that it cannot explain inconsistencies between men and women. One thing that prevents Guisinger’s AFFH
from being entirely plausible is that it cannot explain why AN is far more
prevalent in women. If her hypothesis applies, then would AN not affect both
sexes equally.
AO2/IDA
Another criticism of the explanation is to how the symptoms of AN might be passed on
by natural selection, particularly as they decrease fertility and could even
kill the individual with this condition, this questions the validity of the
explanation.
The theory is also highly speculative and
cannot be falsified. This decreases the validity of the explanation as its not
based on empirical scientific evidence, unlike other theories such as
socialcultural explanations such as the influence of media which is supported
by evidence such as Becker et al, thus making the theory more valid than the
evolutionary explanation.
Researchers such as Holland et al. (1984), however,
have claimed that genetics are the best biological explanation of AN. Research
into genetics’ role tends to come in the form of twin studies. Holland et
al.’s is perhaps the most notable. They researched the genetic link by
comparing MZ and DZ twins, where one twin of each pair was suffering from AN.
They hypothesized that as MZ twins are genetically identical, if AN
characterised entirely genetic, it should be a shared trait. Holland et al.
found a concordance rate of 55% for MZ twins and of only 7% for DZ twins. The
significantly higher concordance rate for MZ twins suggests a genetic
involvement in anorexia nervosa, but clearly it is not the only component in
place. If it was entirely genetic, the concordance rate would surely have been
100%.
AO2
However, Holland et al’s research
has been supported by other research. Kendler et al. (1991) found
similar results in their twin study. Further research has found genetic
contribution to anorexia nervosa is between 50-80% (Klump et al., 2001;
Bulik et al., 2006).
There is gender bias in a lot of the research
carried out into eating disorders as most of the research has concentrated on
the study of women. However according to recent studies, 25% of men suffer from
an eating disorder. This demonstrates that anorexia is not exclusively and
female problem.
A criticism of this theory is that it is
highly deterministic and claims that our behaviour is predetermined by our
genes, in this case the theory is suggesting that anorexia is caused by our
genetics and people who share genetics with someone with anorexia is already
predisposed to the condition. However this ignores free will in which people
can choose how they want to eat, in which it largely agreed that anorexia is a
psychological condition and can be better explained by media as an influence
for anorexia.
No comments:
Post a Comment