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AO1
Many psychologists propose
psychological explanations for schizophrenia. One of these explanations is that
the disorder may be caused by a cognitive deficit. This theory explains
schizophrenia as attention impairment, suggesting that it is caused by a defect
in the patient’s ability to filter incoming information. This means that they
are inundated by external stimuli, which they interpret
incorrectly and this
therefore leads to schizophrenic behavior. Frith suggests that in particular,
people with schizophrenia are unable to distinguish between actions that are
brought about by external forces and those that are generated internally. This
occurs because schizophrenics have problems with self-monitoring and so fail to
keep track of their own intentions. As a result they may mistakenly regard
their own thoughts as alien and as having come from someone else leading to
auditory hallucination. Frith suggests that a failure in self-monitoring could
also explain why schizophrenics may think that their movements are being
controlled externally. Frith also proposes that schizophrenics may not have a
‘Theory of Mind’, the ability to understand what other people’s thoughts or
intentions may be. This could help explain delusions and paranoid symptoms.
AO2
There is some research evidence to
support Frith’s theory that schizophrenia is caused by faulty cognitive
processes. McGuigan (1966) found that the larynx of patients with schizophrenia
was often active during the time they claimed to be experiencing auditory
hallucinations. This suggests that they mistook their own inner speech for that
of someone else and therefore explains why a schizophrenic may claim to hear
voices. However, there is evidence to suggest that explaining schizophrenia in
terms of cognitive functioning may be incorrect. Research has shown that the
cognitive deficit may have a genetic origin and this therefore suggests that
the underlying cause of schizophrenia may be genetic rather than cognitive.
(Faraone et al 1999)
A major criticism of the theory however is the fact that it
is difficult to establish cause and effect. For example in terms of the cognitive
deficit, it is unclear as to whether having cognitive impairments will lead to
schizophrenic symptoms such as delusions and hallucinations or whether having
such symptoms may cause an interference with cognitive functioning. A better
way to establish cause and effect may be to establish what genes are
responsible for the disorder.
IDA
A further criticism of the theory is that it cannot account
for all the symptoms of schizophrenia as it only explains positive symptoms,
suggesting that hallucinations and delusions are brought about by problems in
the cognitive processes, however it cannot explains to negative symptoms such
as loss of emotion, and social withdrawal. These however may be better
explained by family explanations such as
the role of expressed emotion.
Schizophrenia may be explained by looking at family
relationships and the family variables involved such as expressed emotion (EE).
One for example is the “double-bind theory” in which Bateson et al suggest that
children who frequently receive contradictory messages from their parents are
more likely to develop schizophrenia. For example if a mother tells her son
that she love him yet at the same time turns her head away in disgust, the
child receives two conflicting messages about their relationship on different
communicative levels, one of affection of the verbal level, but one of
animosity on the non verbal level. Bateson et al explains that these
interactions prevent the development of an internally coherent construction of
reality, and in the long run manifests it self as schizophrenic symptoms. These
ideas were echoed in the work of psychiatrist R.D Laing.
Another family variable associated with Sz is expressed
emotion. Expressed emotion is a family communication style that involves,
criticism, hostility and emotional over involvement. High levels of EE are most
likely to influence relapse rates. Linszen et al said that schizophrenic
patients are 4 times more likely to relapse returning home to a high EE family,
than a patient returning home to a low EE family. In a study of the relapse
rates among schizophrenics in Iran, found that the high prevalence of EE
in Iranian culture (overprotective
mothers and rejective fathers) was one of the main causes of schizophrenic
relapses. It appears that the negative
emotional climate in these families arouses the patient and leads to stress
beyond his or her already impaired coping mechanisms thus triggering
schizophrenic episode
There is some research evidence to support the double bind
theory, from Beger who found that schizophrenics reported a higher recall of
double bind statements by their mothers than non-schizophrenics. However this
evidence may not be reliable as patients recall may be affected by their
schizophrenia.
AO2
However there are some less supportive studies from Liem
who measured patterns of parental communication in families with a schizophrenic
child and found no difference when compared to normal families. Further more
Hall and Levin analysed data from various previous studies and found no
difference between families with and without a schizophrenic member in the
degree to which verbal and non-verbal communication were in agreement.
A major criticism of the family relationships theory is
that the
theory is based on methodologically flawed studies, for example they did not
include control groups and used a poor operationalized definition of
schizophrenia. Along with this a major problem was that families were studied
retrospectively long after the person’s mental disorder may have affected the
family system.
IDA
There are some ethical issues with the theory also, as the
double bind theory explains that because of mixed messaged parenting per se, it
is the cause of schizophrenia within their child. This is highly unhelpful and
destructive, as it places too much responsibility or blame on family members,
who already have a hard time coping with the situation.
IDA
Expressed emotion as an explanation is considered both a
strength and a weakness. Firstly a weakness because expressed emotion is
clearly not an explanation for the cause of schizophrenia however a strength is
that it’s a well accepted maintenance model for schizophrenia and many prospective studies have been
conducted which support EE hypothesis across many countries and culture
(Miklowitz). So well accepted has the model become that treatment programs for
schizophrenia usually include education and training for family member in
controlling level of EE. Interestingly relapse rates are lower in countries
like Nigeria and India, where there is less of a stigma for schizophrenia and
so strong support is shown consequently exhibiting lower levels of negative
expressed emotion.
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